However, new research is showing us what happens in the brain at the genetic level when the brain is mildly injured many times or severely injured one time to cause neurological deterioration over time and the eventual development of dementia.Continue reading →
Muhammad Ali is remembered as one of the greatest boxers of all time. His physical strength and abilities, his agile footwork in the ring, and his witty and intelligent – and sometimes boastful – running commentaries about himself and his opponents made Ali compelling and appealing to a much wider segment of the population than just those who liked to watch boxing.
But as Ishmael Reed so poignantly points out in his New York Times article about Ali, none of this came without a cost. A very high cost. An eventually fatal cost.Continue reading →
We pause on Memorial Day 2016 (in the United States) to remember our deceased military veterans.
I also pause to remember all those who have died – especially the civilians who weren’t drafted or who didn’t volunteer, but who were simply in the wrong place at the wrong time and became collateral damage – because of war because they should not be forgotten either.
However, in light of Memorial Day, it seems fitting that we should also consider how military service and war increase the risks of developing dementia for veterans.Continue reading →
Today, February 7, 2016, the 50th Super Bowl game in NFL (National Football League) history will be played by the Carolina Panthers and the Denver Broncos. Therefore, it is appropriate to discuss the other dark and dangerous side of this football game – and all the ones played before and all the ones that will be played after – before it is played.
Super Bowl games have become extravagant and lavish productions in the last ten years or so, intended to bring into the audience people who normally either don’t have interest in the game or don’t normally watch football. Super Bowl games also represent an obscenely huge financial windfall for the NFL and for advertisers with enough money to pay for the coveted and outrageously expensive advertising spots during the game.Continue reading →
Our brains are very soft organs that are surrounded by spinal fluid and are protected by the hard outer covering of our skulls.
Under normal circumstances, spinal fluid cushions the brain and keeps it from crashing into the skull. However, if our heads or our bodies are hit hard, our brains can slam into our skulls and result in traumatic brain injuries (TBIs). TBIs are also caused when the skull is fractured and the brain is directly damaged by outside force.
Although concussions, which we’ll discuss later, are sometimes referred to as mild TBIs, the reality is that no injury to the brain is mild and repeated injuries will lead to neurological degeneration that includes dementia.
TBIs are complex neurological injuries that result in a wide variety and severity of symptoms and disabilities.
The least severe symptoms of TBIs – and these may not happen immediately and, in fact, may occur some time after the injury, can include:
Temporary loss of consciousness
Temporary memory loss
Grogginess and sleepiness
Double vision or blurred vision
Nausea or vomiting
Sensitivity to light
Slow reaction to stimuli
The most severe symptoms of TBIs can include:
Extended loss of consciousness or coma
Permanent and severe brain damage
Partial or complete motor paralysis
The most common causes of TBIs, according to the Centers for Disease Control, are:
Car accidents (14.3%)
Head/body collisions with people or things (15.5%)
In the category of TBIs from falling, most of the falls occur disproportionately in the very young (55% of falls among children occur in children between the ages of 0 and 14) and the very old (81% of falls among adults occur in adults who are 65 or older).
Most of the TBIs in the Other category (19%) are from personal firearms and military weapons.
A type of TBI that is more frequently in the headlines today is Chronic Traumatic Encephalopathy (CTE). CTE is brain damage that occurs as a result of repeated concussions (a concussion is defined as injury to the brain from a direct blow to the head or from the head or upper body being violently shaken).
The first identified variant of CTE was described in 1928 by forensic pathologist Dr. Harrison Stanford Martland as pugilistic (from the Latin word pugil, which is translated as “boxer” or “fighter”) dementia. The symptoms included tremors (Parkinsonism), slowed movement, mental confusion, and speech difficulties.
In 1973, the neuropathology of pugilistic dementia was discovered and described by a team of pathologists led by J. A. Corsellis who documented their findings after performing thorough autopsies on the brains of 15 deceased boxers.
Former boxing heavyweight champion Muhammad Ali began boxing in Kentucky when he was 12 years old.
By the age of 18, he had boxed his way to the heavyweight gold medal at the Olympics (1960).
A few months later Ali began his professional boxing career. He quickly gained national prominence because of his skill in the ring and his trademark quote: “Float like a butterfly, sting like a bee. The hands can’t hit what the eyes can’t see.” He boxed professionally until his retirement in 1981.
In 1984, Ali was diagnosed with Parkinsonism (the tremors of pugilistic dementia) and his neurological health has deteriorated steadily to include all the advanced symptoms of this variant of CTE.
His wife, Lonnie, is his caregiver and contributed to a moving article that AARP published last year about what she and Ali deal with on a daily basis as a result of the neurological degeneration that CTE has caused.
CTE has increasingly become a major health concern in the high-contact sports of professional wrestling, ice hockey, soccer, and football as more and more current and retired athletes are showing symptoms consistent with CTE.
In recent years, football – and especially professional football – has become the focal point for a closer examination of CTE. Not only has this sport become more violent in terms of how the game is played, but how concussions are treated – or not treated – has also come under greater scrutiny.
A class action lawsuit has been filed – and a tentative agreement reached with the NFL – by retired NFL football players and/or their families (some of the players have already died from neurodegenerative causes) which claims that players were not (a) adequately protected from suffering concussions, (b) medically treated properly following concussions, and (c) provided adequate medical compensation to treat the burgeoning costs of CTE as it progresses.
This gist of this lawsuit is that the NFL used – and abused – these players to fabulously guild the seemingly-endless coffers of the NFL, often forcing the players by intimidation or fear to get back on the field as soon as they could after suffering a concussion (often in the same game), and then abandoned their responsibility to their former employees (as part of their contractual agreement) as soon as the employees began costing them money instead of making them money.
If there is a silver lining in all of this, it is that the younger NFL players have a much greater awareness of the relationship – and their increased risk – between professional football and CTE.
They are aware of the very real probability that they will be one of the 1 out of every 3 players who develops CTE.
And they’re choosing their long-term health, including their brain health, over temporary fame and fortune.
An unprecedented number of younger – and in-their-prime in the professional football world – NFL players have already retired before the 2015-2016 season begins.
Cortland Finnegan – Age 31
Jake Locker – Age 26
Jason Worilds – Age 27
Chris Borland – Age 24
While Finnegan, Locker, and Worilds did not publicly cite CTE as a factor in their premature retirements from the NFL, there can be no logical reason to doubt that the mounting evidence was a factor in their decisions.
Borland just finished his rookie season (2014-2015) with the San Francisco 49ers, but he revealed after the season that he suffered a concussion in training camp last fall. Instead of reporting the concussion, Borland covered it up so that he could continue to practice and win a starting position on the team.
This is the kind of competitive pressure that gets put on these young players by the NFL (yes, Borland made the decision and he bears the responsibility for it, but had he reported the concussion, he would have been replaced and lost the starting spot and may not have played all season).
Fortunately, though, Borland came to his senses and realized how much he had jeopardized – and would continue to – his neurological health.
As he said on the March 16, 2015 edition of ESPN’s Outside the Lines, “”I just thought to myself, ‘What am I doing? Is this how I’m going to live my adult life, banging my head, especially with what I’ve learned and knew about the dangers?'”
We can only hope that more athletes in high-contact sports will know the higher risks of TBIs they face, not just in the professional leagues, but at the amateur levels, and they will choose to walk away from certain neurological damage.
In the meantime, we have a better understanding just in our daily lives of how TBIs can happen and what the results can be, so I hope that we’re a little more observant and attentive after falls with our little loved ones and our older loved ones, especially those already going through the journey through dementias and Alzheimer’s Disease, as they are even more prone to falling than the general elderly population.
As Sergeant Phil Esterhaus says at the end of every roll call on Hill Street Blues (a favorite TV show of mine during my high school and college years), “Hey, let’s be careful out there.”